Tumor suppressive Ca2+ signaling is driven by IP3 receptor fitness

Authors:

Geert Bultynck1 and Michelangelo Campanella2,3
PDF Download PubMed Link
Affiliations:

1 KU Leuven, Lab. Molecular and Cellular Signaling, Dep. Cellular and Molecular Medicine and Leuven Kanker Instituut (LKI), Campus Gasthuisberg O/N-I bus 802, Herestraat 49, BE-3000 Leuven, Belgium.

2 Department of Comparative Biomedical Sciences, Royal Veterinary College, NW1 0TU, London, United Kingdom.

3 University College London Consortium for Mitochondrial Research, University College London, WC1 6BT, London, United Kingdom.

Keywords: 

cancer, oncogenesis, tumor suppression, IP3R3, calcium signaling, PTEN, FBXL2, BAP1.

Corresponding Author(s):

Geert Bultynck, KU Leuven, Lab. Molecular and Cellular Signaling, Dep. Cellular and Molecular Medicine and Leuven Kanker Instituut (LKI), Campus Gasthuisberg O/N-I bus 802, Herestraat 49, BE-3000 Leuven, Belgium geert.bultynck@kuleuven.be Michelangelo Campanella, Hobday Building, RVC Camden Campus, Royal College Street, NW1 0TU, London, United Kingdom mcampanella@rvc.ac.uk

Conflict of interest statement:

The authors declare no conflict of interest.

Please cite this article as:

Geert Bultynck and Michelangelo Campanella (2017). Tumor suppressive Ca2+ signaling is driven by IP3 receptor fitness. Cell Stress 1(2): 73-78. doi: 10.15698/cst2017.11.109

© 2017 Bultynck and Campanella. This is an open-access article released under the terms of the Creative Commons Attribution (CC BY) license, which allows the unrestricted use, distribution, and reproduction in any medium, provided the original author and source are acknowledged.

Abstract: